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These results reveal an actionable pathway of nutrient acquisition in cancer with direct therapeutic implications

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found that amplifying hypoxia-inducible factor (HIF) signaling pathways triggered ferroptosis in chemotherapy-resistant GBM cells and suppressed tumor growth in animal models, suggesting that inducing ferroptosis by targeting HIF- could improve GBM treatment

Researchers who are not using insulin and who have normal glucose regulation do not face severe hypoglycemia risk from this combination, because both compounds are largely glucose-dependent in their insulin-stimulating effects